STAT3α interacts with nuclear GSK3beta and cytoplasmic RISK pathway and stabilizes rhythm in the anoxic-reoxygenated embryonic heart
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology
Link
http://link.springer.com/content/pdf/10.1007/s00395-011-0152-5.pdf
Reference56 articles.
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2. Boengler K, Heusch G, Schulz R (2010) Mitochondria in postconditioning. Antioxid Redox Signal 2010 Sep 29. [epub ahead of print] doi: 10.1089/ars.2010.3309
3. Boengler K, Hilfiker-Kleiner D, Drexler H, Heusch G, Schulz R (2008) The myocardial JAK/STAT pathway: from protection to failure. Pharmacol Ther 120:172–185. doi: 10.1016/j.pharmthera.2008.08.002
4. Boengler K, Hilfiker-Kleiner D, Heusch G, Schulz R (2010) Inhibition of permeability transition pore opening by mitochondrial STAT3 and its role in myocardial ischemia/reperfusion. Basic Res Cardiol 105:771–785. doi: 10.1007/s00395-010-0124-1
5. Bolli R, Dawn B, Xuan YT (2003) Role of the JAK-STAT pathway in protection against myocardial ischemia/reperfusion injury. Trends Cardiovasc Med 13:72–79. doi: S105017380200230X
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