Exogenous GDF11 induces cardiac and skeletal muscle dysfunction and wasting-Reference-Cited by-同舟云学术

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Exogenous GDF11 induces cardiac and skeletal muscle dysfunction and wasting

Published:2017-06-24 Issue:4 Volume:112 Page:
ISSN:0300-8428
Container-title:Basic Research in Cardiology
language:en
Short-container-title:Basic Res Cardiol

Author:

Zimmers Teresa A.,Jiang Yanling,Wang Meijing,Liang Tiffany W.,Rupert Joseph E.,Au Ernie D.,Marino Francesco E.,Couch Marion E.,Koniaris Leonidas G.

Funder

Lilly Foundation

National Institute of Diabetes and Digestive and Kidney Diseases

National Cancer Institute

Lustgarten Foundation (US)

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://link.springer.com/article/10.1007/s00395-017-0639-9/fulltext.html

Reference35 articles.

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3. Bodine SC, Latres E, Baumhueter S, Lai VK, Nunez L, Clarke BA, Poueymirou WT, Panaro FJ, Na E, Dharmarajan K, Pan ZQ, Valenzuela DM, DeChiara TM, Stitt TN, Yancopoulos GD, Glass DJ (2001) Identification of ubiquitin ligases required for skeletal muscle atrophy. Science (New York, NY) 294:1704–1708. doi: 10.1126/science.1065874

4. Bonetto A, Aydogdu T, Jin X, Zhang Z, Zhan R, Puzis L, Koniaris LG, Zimmers TA (2012) JAK/STAT3 pathway inhibition blocks skeletal muscle wasting downstream of IL-6 and in experimental cancer cachexia. Am J Physiol Endocrinol Metab 303:E410–E421. doi: 10.1152/ajpendo.00039.2012

5. Dirkx E, da Costa Martins PA, De Windt LJ (2013) Regulation of fetal gene expression in heart failure. Biochim Biophys Acta 1832:2414–2424. doi: 10.1016/j.bbadis.2013.07.023

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