Human iPS cell model of type 3 long QT syndrome recapitulates drug-based phenotype correction-Reference-Cited by-同舟云学术

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Human iPS cell model of type 3 long QT syndrome recapitulates drug-based phenotype correction

Published:2016-01-23 Issue:2 Volume:111 Page:
ISSN:0300-8428
Container-title:Basic Research in Cardiology
language:en
Short-container-title:Basic Res Cardiol

Author:

Malan Daniela,Zhang Miao,Stallmeyer Birgit,Müller Jovanca,Fleischmann Bernd K.,Schulze-Bahr Eric,Sasse Philipp,Greber Boris

Funder

European Union / State of North Rhine Westfalia

Chemical Genomics Centre of the Max Planck Society

Bundesinstitut für Risikobewertung

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://link.springer.com/content/pdf/10.1007/s00395-016-0530-0.pdf

Reference34 articles.

1. Antzelevitch C, Shimizu W (2002) Cellular mechanisms underlying the long QT syndrome. Curr Opin Cardiol 17:43–51

2. Balser JR (2001) The cardiac sodium channel: gating function and molecular pharmacology. J Mol Cell Cardiol 33:599–613. doi: 10.1006/jmcc.2000.1346

3. Belardinelli L, Shryock JC, Fraser H (2006) Inhibition of the late sodium current as a potential cardioprotective principle: effects of the late sodium current inhibitor ranolazine. Heart 92(Suppl 4):iv6–iv14. doi: 10.1136/hrt.2005.078790

4. Bennett PB, Yazawa K, Makita N, George AL Jr (1995) Molecular mechanism for an inherited cardiac arrhythmia. Nature 376:683–685. doi: 10.1038/376683a0

5. Crotti L, Celano G, Dagradi F, Schwartz PJ (2008) Congenital long QT syndrome. Orphanet J Rare Dis 3:18. doi: 10.1186/1750-1172-3-18

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