Deletion of newly described pro-survival molecule Pellino-1 increases oxidative stress, downregulates cIAP2/NF-κB cell survival pathway, reduces angiogenic response, and thereby aggravates tissue function in mouse ischemic models-Reference-Cited by-同舟云学术

Deletion of newly described pro-survival molecule Pellino-1 increases oxidative stress, downregulates cIAP2/NF-κB cell survival pathway, reduces angiogenic response, and thereby aggravates tissue function in mouse ischemic models

Published:2020-06-14 Issue:4 Volume:115 Page:
ISSN:0300-8428
Container-title:Basic Research in Cardiology
language:en
Short-container-title:Basic Res Cardiol

Author:

Selvaraju Vaithinathan,Thirunavukkarasu Mahesh,Joshi Mandip,Oriowo Babatunde,Shaikh Inam A.,Rishi Muhammad Tipu,Tapias Leonidas,Coca-Soliz Vladimir,Saad Ibnalwalid,Campbell Jacob,Pradeep Seetur R.,Swaminathan Santosh,Yee Siu-Pok,McFadden David W.,Alexander Palesty J.,Maulik Nilanjana

Funder

National Institute of Health

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

https://link.springer.com/content/pdf/10.1007/s00395-020-0804-4.pdf

Reference74 articles.

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2. Adluri RS, Thirunavukkarasu M, Zhan L, Akita Y, Samuel SM, Otani H, Ho YS, Maulik G, Maulik N (2011) Thioredoxin 1 enhances neovascularization and reduces ventricular remodeling during chronic myocardial infarction: a study using thioredoxin 1 transgenic mice. J Mol Cell Cardiol 50:239–247. https://doi.org/10.1016/j.yjmcc.2010.11.002

3. Afzal MR, Haider H, Idris NM, Jiang S, Ahmed RP, Ashraf M (2010) Preconditioning promotes survival and angiomyogenic potential of mesenchymal stem cells in the infarcted heart via NF-kappaB signaling. Antioxid Redox Signal 12:693–702. https://doi.org/10.1089/ars.2009.2755