The temporal relationship between p38 MAPK and HSP27 activation in ischaemic and pharmacological preconditioning-Reference-Cited by-同舟云学术

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The temporal relationship between p38 MAPK and HSP27 activation in ischaemic and pharmacological preconditioning

Published:2004-11-03 Issue:1 Volume:100 Page:35-47
ISSN:0300-8428
Container-title:Basic Research in Cardiology
language:en
Short-container-title:Basic Res Cardiol

Author:

Marais Erna,Genade Sonia,Salie Ruduwaan,Huisamen Barbara,Maritz Stefan,Moolman Johan A.,Lochner Amanda

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://link.springer.com/content/pdf/10.1007/s00395-004-0495-7.pdf

Reference55 articles.

1. Armstrong SC, Delacey MH, Ganote CE (1999) Phosphorylation state of hsp27 and p38 MAPK during preconditioning and protein phosphatase inhibitor protection of rabbit cardiomyocytes. J Mol Cell Cardiol 31:555?567

2. Baines CP, Pass JM, Ping P (2001) Protein kinases and kinase-modulated effectors in the late phase of ischemic preconditioning. Basic Res Cardiol 96:207?218

3. Behrends M, Schulz R, Post H, Alexandrov A, Belosjorow S, Michel MC, Heusch G (2000) Inconsistent relation of MAPK activation to infarct size reduction by ischemic preconditioning in pigs. Am J Physiol Heart Circ Physiol 279:H1111?1119

4. Benjamin IJ, McMillan DR (1998) Stress (heat shock) proteins: molecular chaperones in cardiovascular biology and disease. Circ Res 83:117?132

5. Bogoyevitch MA, Gillespie-Brown J, Ketterman AJ, Fuller SJ, Ben-Levy R, Ashworth A, Marshall CJ, Sugden PH (1996) Stimulation of the stress-activated mitogen-activated protein kinase subfamily in the perfused rat heart. p38/ERK mitogen-activated protein kinases and c-Jun N-terminal kinases are activated by ischemia/reperfusion. Circ Res 79:162?173

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