Oxidized low-density lipoprotein (oxLDL) affects load-free cell shortening of cardiomyocytes in a proprotein convertase subtilisin/kexin 9 (PCSK9)-dependent way
Author:
Funder
Sanofi
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology
Link
http://link.springer.com/article/10.1007/s00395-017-0650-1/fulltext.html
Reference36 articles.
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2. Chandrakala AN, Sukul D, Selvarajan K, Sai-Sudhakar C, Sun B, Parthasarathy S (2012) Induction of brain natriuretic peptide and monocyte chemotactic protein-1 gene expression by oxidized low-density lipoprotein: relevance of ischemic heart failure. Am J Physiol Cell Physiol 302:C165–C177. doi: 10.1152/ajpcell/00116.2011
3. Ding Z, Liu S, Wang X, Deng X, Fan Y, Shahanawaz J, Shmookler Reis RJ, Varughese KI, Sawamura T, Mehta JL (2015) Cross-talk between LOX-1 and PCSK9 in vascular tissue. Cardiovasc Res 107:556–567. doi: 10.1093/cvr/cvv178
4. Euler G (2016) Mechanisms of cardiac cell death. In: Schlüter K-D (ed) Cardiomyocytes—active players in cardiac disease. Springer, Berlin, pp 247–266. doi: 10.1007/978-3-319-31251-4_9
5. Fearon IM (2006) OxLDL enhances L-type Ca2+ currents via lysophosphatidylcholine-induced mitochondrial reactive oxygen species (ROS) production. Cardiovasc Res 69:855–864. doi: 10.1016/j.cardiores.2005.11.019
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