Reduction of no-reflow and reperfusion injury with the synthetic 17β-aminoestrogen compound Prolame is associated with PI3K/Akt/eNOS signaling cascade

Author:

Hernández-Reséndiz Sauri,Palma-Flores Carlos,De los Santos Sergio,Román-Anguiano Nadia G.,Flores Mirthala,de la Peña Aurora,Flores Pedro L.,Fernández-G Juan M.,Coral-Vázquez Ramón M.,Zazueta Cecilia

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Reference52 articles.

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2. Beckman JS, Beckman TW, Chen J, Marshall PA, Freeman BA (1990) Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide. Proc Natl Acad Sci USA 87:1620–1624. doi: 10.1073/pnas.87.4.1620

3. Brady AJ, Warren JB, Poole-Wilson PA, Williams TJ, Harding SE (1993) Nitric oxide attenuates cardiac myocyte contraction. Am J Physiol 265:H176–H182

4. Butler MJ, ChanW Taylor AJ, Dart AM, Duffy SJ (2011) Management of the no-reflow phenomenon. Pharmacol Ther 132:72–85. doi: 10.1016/j.pharmthera.2011.05.010

5. Coral-Vazquez RM, Cohn RD, Moore SA, Hill JA, Weiss RM, Davisson RL, Straub V, Barresi R, Bansal D, Hrstka RF, Williamson R, Campbell KP (1999) Disruption of the sarcoglycan-sarcospan complex in vascular smooth muscle: a novel mechanism for cardiomyopathy and muscular dystrophy. Cell 98:465–474. doi: 10.1016/S0092-8674(00)81975-3

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