Indices of Metabolic Dysfunction and Oxidative Stress-Reference-Cited by-同舟云学术

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Indices of Metabolic Dysfunction and Oxidative Stress

Published:2007-03-07 Issue:4-5 Volume:32 Page:717-722
ISSN:0364-3190
Container-title:Neurochemical Research
language:en
Short-container-title:Neurochem Res

Author:

Casadesus Gemma,Moreira Paula I.,Nunomura Akihiko,Siedlak Sandra L.,Bligh-Glover William,Balraj Elizabeth,Petot Grace,Smith Mark A.,Perry George

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Medicine,Biochemistry

Link

http://link.springer.com/content/pdf/10.1007/s11064-007-9296-y.pdf

Reference56 articles.

1. Hardy J, Selkoe DJ (2002) The amyloid hypothesis of Alzheimer’s disease: progress and problems on the road to therapeutics. Science 297:353–356

2. Hardy JA, Higgins GA (1992) Alzheimer’s disease: the amyloid cascade hypothesis. Science 256:184–185

3. Selkoe DJ (2001) Alzheimer’s disease results from the cerebral accumulation and cytotoxicity of amyloid beta-protein. J Alzheimers Dis 3:75–80

4. Trojanowski JQ, Clark CM, Arai H, Lee VM (1999) Elevated levels of tau in cerebrospinal fluid: implications for the antemortem diagnosis of Alzheimer’s disease. J Alzheimers Dis 1:297–305

5. Gomez-Ramos A, Smith MA, Perry G, Avila J (2004) Tau phosphorylation and assembly. Acta Neurobiol Exp (Wars) 64:33–39

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1. Chronic hyperglycemia impairs hippocampal neurogenesis and memory in an Alzheimer’s disease mouse model;Neurobiology of Aging;2020-08

2. O-GlcNAcylation of Amyloid-β Protein Precursor by Insulin Signaling Reduces Amyloid-β Production;Journal of Alzheimer's Disease;2019-06-18

3. Evaluation of Metabolic and Synaptic Dysfunction Hypotheses of Alzheimer's Disease (AD): A Meta-Analysis of CSF Markers;Current Alzheimer Research;2018-01-03

4. O-GlcNAcylation of amyloid-β precursor protein at threonine 576 residue regulates trafficking and processing;Biochemical and Biophysical Research Communications;2017-08

5. Effects of caloric restriction on O-GlcNAcylation, Ca2+ signaling, and learning impairment in the hippocampus of ob/ob mice;Neurobiology of Aging;2016-08

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