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Propofol Inhibits Ferroptotic Cell Death Through the Nrf2/Gpx4 Signaling Pathway in the Mouse Model of Cerebral Ischemia–Reperfusion Injury

  • Published:2022-11-19 Issue:3 Volume:48 Page:956-966
  • ISSN:0364-3190
  • Container-title:Neurochemical Research
  • language:en
  • Short-container-title:Neurochem Res

Author:

Fan Gui-bo,Li Yan,Xu Gao-shuo,Zhao A.-yang,Jin Hong-jiang,Sun Si-qi,Qi Si-hua

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Medicine,Biochemistry

Reference52 articles.

1. Benjamin EJ, Muntner P, Alonso A, Bittencourt MS, Callaway CW, Carson AP, Chamberlain AM, Chang AR, Cheng S, Das SR et al (2019) Heart disease and stroke statistics-2019 update: a report from the American Heart Association. Circulation 139:e56–e528. https://doi.org/10.1161/CIR.0000000000000659

2. Burda J, Danielisova V, Nemethova M, Gottlieb M, Matiasova M, Domorakova I, Mechirova E, Ferikova M, Salinas M, Burda R (2006) Delayed postconditionig initiates additive mechanism necessary for survival of selectively vulnerable neurons after transient ischemia in rat brain. Cell Mol Neurobiol 26:1141–1151. https://doi.org/10.1007/s10571-006-9036-x

3. Lee TK, Lee JC, Kim DW, Kim B, Sim H, Kim JD, Ahn JH, Park JH, Lee CH, Won MH et al (2021) Ischemia-reperfusion under hyperthermia increases heme oxygenase-1 in pyramidal neurons and astrocytes with accelerating neuronal loss in gerbil hippocampus. Int J Mol Sci 22:3963. https://doi.org/10.3390/ijms22083963

4. Lai Z, Zhang L, Su J, Cai D, Xu Q (2016) Sevoflurane postconditioning improves long-term learning and memory of neonatal hypoxia-ischemia brain damage rats via the PI3K/Akt-mPTP pathway. Brain Res 1630:25–37. https://doi.org/10.1016/j.brainres.2015.10.050

5. Galluzzi L, Vitale I, Aaronson SA, Abrams JM, Adam D, Agostinis P, Alnemri ES, Altucci L, Amelio I, Andrews DW et al (2018) Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018. Cell Death Differ 25:486–541. https://doi.org/10.1038/s41418-017-0012-4

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