Silencing of UCA1 Protects Against MPP+-Induced Cytotoxicity in SK-N-SH Cells via Modulating KCTD20 Expression by Sponging miR-423-5p-Reference-Cited by-同舟云学术

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Silencing of UCA1 Protects Against MPP+-Induced Cytotoxicity in SK-N-SH Cells via Modulating KCTD20 Expression by Sponging miR-423-5p

Published:2021-01-19 Issue:4 Volume:46 Page:878-887
ISSN:0364-3190
Container-title:Neurochemical Research
language:en
Short-container-title:Neurochem Res

Author:

Zheng Yanhua,Liu Junpeng,Zhuang Jiajun,Dong Xiaoyan,Yu Miao,Li Zhihui

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Medicine,Biochemistry

Link

http://link.springer.com/content/pdf/10.1007/s11064-020-03214-9.pdf

Reference36 articles.

1. Beitz JM (2014) Parkinson’s disease: a review. Front Biosci 6:65–74

2. Abeliovich A, Gitler AD (2016) Defects in trafficking bridge Parkinson’s disease pathology and genetics. Nature 539:207–216

3. Kalia LV, Kalia SK, Lang AE (2015) Disease-modifying strategies for Parkinson’s disease. Mov Disord 30:1442–1450

4. Waldthaler J, Timmermann L (2019) Update on diagnostics and therapy of idiopathic Parkinson’s disease. Fortschr Neurol Psychiatr 87:445–461

5. Lekka E, Hall J (2018) Noncoding RNAs in disease. FEBS Lett 592:2884–2900

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2. Diagnostic value and cognitive regulatory roles of long non-coding RNA UCA1 in Alzheimer’s disease;Neuroscience Letters;2024-04

3. Competing endogenous RNA (ceRNA) networks in Parkinson's disease: A systematic review;Frontiers in Cellular Neuroscience;2023-01-24

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5. Trophoblast Exosomal UCA1 Induces Endothelial Injury through the PFN1-RhoA/ROCK Pathway in Preeclampsia: A Human-Specific Adaptive Pathogenic Mechanism;Oxidative Medicine and Cellular Longevity;2022-09-15

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