Endogenous CBS–H2S Pathway Contributes to the Development of CCI-Induced Neuropathic Pain-Reference-Cited by-同舟云学术

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Endogenous CBS–H2S Pathway Contributes to the Development of CCI-Induced Neuropathic Pain

Published:2016-03-09 Issue:6 Volume:41 Page:1381-1389
ISSN:0364-3190
Container-title:Neurochemical Research
language:en
Short-container-title:Neurochem Res

Author:

Gui Yulong,Li Aiyuan,Qiu Bihui,Chen Feng,Chen Liang,Liu Daming,Chen Shuxian,Zhou Wei,Zhou Hong

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Medicine,Biochemistry

Link

http://link.springer.com/content/pdf/10.1007/s11064-016-1842-z.pdf

Reference28 articles.

1. Kim HK, Park SK, Zhou JL, Taglialatela G, Chung K, Coggeshall RE, Chung JM (2004) Reactive oxygen species (ROS) play an important role in a rat model of neuropathic pain. Pain 111(1):116–124

2. Schäfers M, Sommer C (2007) Anticytokine therapy in neuropathic pain management. Expert Rev Neurother 7(11):1613–1627

3. Flatters SJ, Bennett GJ (2006) Studies of peripheral sensory nerves in paclitaxel-induced painful peripheral neuropathy: evidence for mitochondrial dysfunction. Pain 122(3):245–257

4. Takahashi T, Aoki Y, Okubo K, Maeda Y, Sekiguchi F, Mitani K, Nishikawa H, Kawabata A (2010) Upregulation of Ca v 3.2 T-type calcium channels targeted by endogenous hydrogen sulfide contributes to maintenance of neuropathic pain. Pain 150(1):183–191

5. Kimura Y, Mikami Y, Osumi K, Tsugane M, Oka JI, Kimura H (2013) Polysulfides are possible H2S-derived signaling molecules in rat brain. FASEB J 27(6):2451–2457

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