Sphingosylphosphorylcholine Attenuated β–Amyloid Production by Reducing BACE1 Expression and Catalysis in PC12 Cells-Reference-Cited by-同舟云学术

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Sphingosylphosphorylcholine Attenuated β–Amyloid Production by Reducing BACE1 Expression and Catalysis in PC12 Cells

Published:2011-06-15 Issue:11 Volume:36 Page:2083-2090
ISSN:0364-3190
Container-title:Neurochemical Research
language:en
Short-container-title:Neurochem Res

Author:

Yi Hyoseok,Lee Seong Jin,Lee Jiyeong,Myung Chang-Seon,Park Woo-Kyu,Lim Hee-Jong,Lee Ge Hyeong,Kong Jae Yang,Cho Heeyeong

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Medicine,Biochemistry

Link

http://link.springer.com/content/pdf/10.1007/s11064-011-0532-0.pdf

Reference47 articles.

1. Haass C, Schlossmacher MG, Hung AY et al (1992) Amyloid beta-peptide is produced by cultured cells during normal metabolism. Nature 359:322–325

2. LaFerla FM, Green KN, Oddo S (2007) Intracellular amyloid-beta in Alzheimer’s disease. Nat Rev Neurosci 8:499–509

3. De Jonghe C, Esselens C, Kumar-Singh S et al (2001) Pathogenic APP mutations near the gamma-secretase cleavage site differentially affect Abeta secretion and APP C-terminal fragment stability. Hum Mol Genet 10:1665–1671

4. Herreman A, Serneels L, Annaert W et al (2000) Total inactivation of gamma-secretase activity in presenilin-deficient embryonic stem cells. Nat Cell Biol 2:461–462

5. Selkoe DJ (1998) The cell biology of beta-amyloid precursor protein and presenilin in Alzheimer’s disease. Trends Cell Biol 8:447–453

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