Comparative Effects of Acute or Chronic Administration of Levodopa to 6-OHDA-lesioned Rats on the Expression and Phosphorylation of N-methyl-d-aspartate Receptor NR1 Subunits in the Striatum
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,General Medicine,Biochemistry
Link
http://link.springer.com/content/pdf/10.1007/s11064-009-9939-2.pdf
Reference32 articles.
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3. Chase TN, Oh JD (2000) Striatal mechanisms and pathogenesis of Parkinsonian signs and motor complications. Ann Neurol 47:S122–S129. doi: 10.1002/1531-8249(200001)47:1<122::AID-ANA21>3.0.CO;2-9
4. Obeso JA, Rodriguez-Oroz MC, Chana P et al (2000) The evolution and origin of motor complications in Parkinson’s disease. Neurology 55:S13–S20
5. Chase TN, Oh JD (2000) Striatal dopamine- and glutamate-mediated dysregulation in experimental parkinsonism. Trends Neurosci 23:S86–S91. doi: 10.1016/S1471-1931(00)00018-5
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1. Modulation of CaMKIIa-GluN2B interaction in levodopa-induced dyskinesia in 6-OHDA-lesioned Parkinson’s rats;Biomedicine & Pharmacotherapy;2018-11
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4. Interactions of CaMKII with dopamine D2 receptors: roles in levodopa-induced dyskinesia in 6-hydroxydopamine lesioned Parkinson's rats;Scientific Reports;2014-10-29
5. Brain Morphometry and the Neurobiology of Levodopa-Induced Dyskinesias: Current Knowledge and Future Potential for Translational Pre-Clinical Neuroimaging Studies;Frontiers in Neurology;2014-06-12
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