NFAT5 Regulated by STUB1, Facilitates Malignant Cell Survival and p38 MAPK Activation by Upregulating AQP5 in Chronic Lymphocytic Leukemia
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics,Molecular Biology,General Medicine,Ecology, Evolution, Behavior and Systematics,Biochemistry
Link
http://link.springer.com/content/pdf/10.1007/s10528-021-10040-3.pdf
Reference31 articles.
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2. Barthel R et al (2020) CD74 is dispensable for development of chronic lymphocytic leukemia in Emicro-TCL1 transgenic mice. Leuk Lymphoma. https://doi.org/10.1080/10428194.2020.1791851
3. Chae YK et al (2008) Human AQP5 plays a role in the progression of chronic myelogenous leukemia (CML). PLoS ONE 3:e2594. https://doi.org/10.1371/journal.pone.0002594
4. Fernandez-Castillo N et al (2015) Transcriptomic and genetic studies identify NFAT5 as a candidate gene for cocaine dependence. Transl Psychiatry 5:e667. https://doi.org/10.1038/tp.2015.158
5. Germann S et al (2012) Dual role of the ddx5/ddx17 RNA helicases in the control of the pro-migratory NFAT5 transcription factor. Oncogene 31:4536–4549. https://doi.org/10.1038/onc.2011.618
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