Alterations in biomechanical properties of the cornea among patients with polycystic kidney disease
Author:
Publisher
Springer Science and Business Media LLC
Subject
Ophthalmology
Link
http://link.springer.com/article/10.1007/s10792-017-0619-y/fulltext.html
Reference36 articles.
1. Harris PC, Torres VE (2009) Polycystic kidney disease. Annu Rev Med 60(1):321–337. doi: 10.1146/annurev.med.60.101707.125712
2. Rossetti S, Harris PC (2007) Genotype–phenotype correlations in autosomal dominant and autosomal recessive polycystic kidney disease. J Am Soc Nephrol 18(5):1374–1380. doi: 10.1681/ASN.2007010125
3. Adeva M, El-Youssef M, Rossetti S, Kamath PS, Kubly V, Consugar MB et al (2006) Clinical and molecular characterization defines a broadened spectrum of autosomal recessive polycystic kidney disease (ARPKD). Medicine (Baltimore) 85(1):1–21. doi: 10.1097/01.md.0000200165.90373.9a
4. Ward CJ, Hogan MC, Rossetti S, Walker D, Sneddon T, Wang X et al (2002) The gene mutated in autosomal recessive polycystic kidney disease encodes a large, receptor-like protein. Nat Genet 30(3):259–269. doi: 10.1038/ng833
5. Yoder BK, Hou X, Guay-Woodford LM (2002) The polycystic kidney disease proteins, polycystin-1, polycystin-2, polaris, and cystin, are co-localized in renal cilia. J Am Soc Nephrol 13(10):2508–2516. doi: 10.1097/01.ASN.0000029587.47950.25
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