Iclaprim activity against wild-type and corresponding thymidine kinase–deficient Staphylococcus aureus in a mouse protection model
Author:
Publisher
Springer Science and Business Media LLC
Subject
Infectious Diseases,Microbiology (medical),General Medicine
Link
http://link.springer.com/article/10.1007/s10096-018-3440-2/fulltext.html
Reference11 articles.
1. Sader HS, Fritsche TR, Jones RN (2009) Potency and bactericidal activity of iclaprim against recent clinical gram-positive isolates. Antimicrob Agents Chemother 53:2171–5
2. Koch AE, Burchall JJ (1971) Reversal of the antimicrobial activity of trimethoprim by thymidine in commercially prepared media. Appl Microbiol 22(5):812–817
3. Nottebrock H, Then R (1977) Thymidine concentrations in serum and urine of different animal species and man. Biochem Pharmacol 26:2175–2179
4. Then R, Angehrn P (1974) The biochemical basis of the antimicrobial action of sulfonamides and trimethoprim in vivo-I. Action of sulfonamides and trimethoprim in blood and urine. Biochem Pharmacol 23:2977–2982
5. Then R, Angehrn P (1975) The biochemical basis of the antimicrobial action of sulfonamides and trimethoprim in vivo-II. Experiments on the limited thymine and thymidine availability in blood and urine. Biochem Pharmacol 24:1003–1006
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1. Iclaprim reduces the incidence and severity of Staphylococcus aureus-induced septic arthritis in a murine model;Access Microbiology;2019-09-01
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