Response of TRPM2 Channel to Hypercapnic Acidosis and Role of Zn, Se, and GSH

Author:

Ergun D. Duzgun,Dursun S.,Ozsobaci N. Pastaci,Naziroglu M.,Ozcelik D.

Funder

Istanbul Üniversitesi

Publisher

Springer Science and Business Media LLC

Subject

Biochemistry, medical,Inorganic Chemistry,Clinical Biochemistry,General Medicine,Biochemistry,Endocrinology, Diabetes and Metabolism

Reference38 articles.

1. Jay B, Dean J (2010) Hypercapnia causes cellular oxidation and nitrosation in addition to acidosis: implications for CO2 chemoreceptor function and dysfunction. Appl Phys 108:1786–1795

2. Matthaeis A, Greco A, Dagostino MP, Paroni G, Fontana A, Vinciguerra M (2014) Effects of hypercapnia on peripheral vascular reactivity in elderly patients with acute exacerbation of chronic obstructive pulmonary disease. Clin Interv Aging 9:871–878

3. Cheng HLM (2012) Effect of hyperoxia and hypercapnia on tissue oxygen and perfusion response in the normal liver and kidney. PLoS One 7(7):e40485

4. Stengl M, Ledvinova L, Chvojka J, Benes J, Jarkovska D, Holas J (2013) Effects of clinically relevant acute hypercapnic and metabolic acidosis on the cardiovascular system: an experimental porcine study. Crit Care 17(6-R303):1–12

5. Imber AN, Putnam RW (2012) Postnatal development and activation of L-type Ca+2 currents in locus ceruleus neurons: implications for a role for Ca+2 in central chemosensitivity. J Appl Physiol 112(10):1715–1726

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