High Copper Levels Promotes Broiler Hepatocyte Mitochondrial Permeability Transition In Vivo and In Vitro-Reference-Cited by-同舟云学术

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High Copper Levels Promotes Broiler Hepatocyte Mitochondrial Permeability Transition In Vivo and In Vitro

Published:2011-04-12 Issue:1-3 Volume:144 Page:636-646
ISSN:0163-4984
Container-title:Biological Trace Element Research
language:en
Short-container-title:Biol Trace Elem Res

Author:

Su Rongsheng,Wang Rongmei,Cao Huabin,Pan Jiaqiang,Chen Lijun,Li Chengmei,Shi Dayou,Tang Zhaoxin

Publisher

Springer Science and Business Media LLC

Subject

Biochemistry, medical,Inorganic Chemistry,Clinical Biochemistry,General Medicine,Biochemistry,Endocrinology, Diabetes and Metabolism

Link

http://link.springer.com/content/pdf/10.1007/s12011-011-9015-z.pdf

Reference29 articles.

1. Mertz W (1987) Trace elements in human and animal nutrition, 5th edn. Academic, Orlando, pp 301–350

2. Chuttani HK, Gupta PS, Gulati S, Gupta DN (1965) Acute copper sulfate poisoning. Am J Med 39:849–854

3. Koizumi T, Yokota T, Shirakura H, Tatsumoto H, Suzuki KT (1994) Potential mechanism of cadmium-induced cytotoxicity in rat hepatocytes: inhibitory action of cadmium on mitochondrial respiratory activity. Toxicol 92:115–125

4. Xiang LX, Shao JZ (2003) Role of intracellular Ca2+, reactive oxygen species, mitochondria transmembrane potential, and antioxidant enzymes in heavy metal-induced apoptosis in fish cell. Bull Environ Contam Toxicol 71:114–122

5. Zhao M, Antunes F, Eaton JW, Brunk UT (2003) Lysosomal enzymes promote mitochondrial oxidant product, cytochrome c release and apoptosis. Eur J Biochem 270:3778–3786

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