Upregulation of host genes during disease progression in bovine leukemia virus infection is independent of overexpression of viral transcriptional regulators in vitro
Author:
Funder
Ministry of Agriculture, Forestry and Fisheries
Publisher
Springer Science and Business Media LLC
Subject
Virology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00705-023-05713-w.pdf
Reference27 articles.
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2. Durkin K, Rosewick N, Artesi M, Hahaut V, Griebel P, Arsic N, Burny A, Georges M, Van den Broeke A (2016) Characterization of novel Bovine Leukemia Virus (BLV) antisense transcripts by deep sequencing reveals constitutive expression in tumors and transcriptional interaction with viral microRNAs. Retrovirology 13(1):33. https://doi.org/10.1186/s12977-016-0267-8
3. Somura Y, Sugiyama E, Fujikawa H, Murakami K (2014) Comparison of the copy numbers of bovine leukemia virus in the lymph nodes of cattle with enzootic bovine leukosis and cattle with latent infection. Arch Virol 159(10):2693–2697. https://doi.org/10.1007/s00705-014-2137-9
4. Ohno A, Takeshima SN, Matsumoto Y, Aida Y (2015) Risk factors associated with increased bovine leukemia virus proviral load in infected cattle in Japan from 2012 to 2014. Virus Res 210:283–290. https://doi.org/10.1016/j.virusres.2015.08.020
5. Pluta A, Jaworski JP, Douville RN (2020) Regulation of expression and latency in BLV and HTLV. Viruses 12(10):1079. https://doi.org/10.3390/v12101079
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1. The bovine leukemia virus-derived long non-coding RNA AS1-S binds to bovine hnRNPM and alters the interaction between hnRNPM and host mRNAs;Microbiology Spectrum;2023-10-17
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