Increased expression of BCL11B and its recruited chromatin remodeling factors during highly active antiretroviral therapy synergistically represses the transcription of human immunodeficiency virus type 1 and is associated with residual immune activation-Reference-Cited by-同舟云学术

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Increased expression of BCL11B and its recruited chromatin remodeling factors during highly active antiretroviral therapy synergistically represses the transcription of human immunodeficiency virus type 1 and is associated with residual immune activation

Published:2019-12-11 Issue:2 Volume:165 Page:321-330
ISSN:0304-8608
Container-title:Archives of Virology
language:en
Short-container-title:Arch Virol

Author:

Wang Juan^ORCID,Yang Zongxing,Wu Nan-Ping,Yang Jin

Funder

the Natural Science Funds of Zhejiang Province

the Medical and Health Science and Technology Planning Project of Zhejiang Province

the Public Technology Research/Social Development Program of Zhejiang Province

the Hangzhou Science and Technology Development Program

Publisher

Springer Science and Business Media LLC

Subject

Virology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00705-019-04475-8.pdf

Reference34 articles.

1. Tyagi M, Bukrinsky M (2012) Human immunodeficiency virus (HIV) latency: the major hurdle in HIV eradication. Mol Med 18:1096–1108

2. Paiardini M, Muller-Trutwin M (2013) HIV-associated chronic immune activation. Immunol Rev 254:78–101

3. Mousseau G, Valente ST (2017) Role of host factors on the regulation of Tat-mediated HIV-1 transcription. Curr Pharm Des 23:4079–4090

4. Avram D, Fields A, Top KPO, Nevrivy DJ, Ishmael JE, Leid M (2000) Isolation of a novel family of C2H2 zinc finger proteins implicated in transcriptional repression mediated by chicken ovalbumin upstream promoter transcription factor (COUP-TF) orphan nuclear receptors. J Biol Chem 275:10315–10322

5. Cysique LA, Juge L, Lennon MJ, Gates TM, Jones SP, Lovelace MD, Rae CD, Johnson TP, Nath A, Brew BJ (2019) HIV brain latency as measured by CSF Bcl11b relates to disrupted brain cellular energy in virally suppressed HIV infection. AIDS 33:433–441

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