A lack of Fas/FasL signalling leads to disturbances in the antiviral response during ectromelia virus infection-Reference-Cited by-同舟云学术

A lack of Fas/FasL signalling leads to disturbances in the antiviral response during ectromelia virus infection

Published:2016-01-18 Issue:4 Volume:161 Page:913-928
ISSN:0304-8608
Container-title:Archives of Virology
language:en
Short-container-title:Arch Virol

Author:

Bień K.,Sobańska Z.,Sokołowska J.,Bąska P.,Nowak Z.,Winnicka A.,Krzyzowska M.

Publisher

Springer Science and Business Media LLC

Subject

Virology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00705-015-2746-y.pdf

Reference50 articles.

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2. Esteban DJR, Buller ML (2005) Ectromelia virus: the causative agent of mousepox. J Gen Virol 861:2645–2659

3. Wallace GD, Buller RM (1985) Kinetics of ectromelia virus (mousepox) transmission and clinical response in C57BL/6j, BALB/cByj and AKR/J inbred mice. Lab Anim Sci 35:41–46

4. Jacoby RO, Bhatt PN, Brownstein DG (1989) Evidence that NK cells and interferon are required for genetic resistance to lethal infection with ectromelia virus. Arch Virol 108:49–58

5. Delano ML, Brownstein DG (1995) Innate resistance to lethal mousepox is genetically linked to the NK gene complex on chromosome 6 and correlates with early restriction of virus replication by cells with an NK phenotype. J Virol 69:5875–5877

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