Classical swine fever virus down-regulates endothelial connexin 43 gap junctions
Author:
Publisher
Springer Science and Business Media LLC
Subject
Virology,General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s00705-010-0693-1.pdf
Reference36 articles.
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2. Bauhofer O, Summerfield A, Sakoda Y, Tratschin J, Hofmann MA, Ruggli N (2007) Classical swine fever virus Npro interacts with interferon regulatory factor 3 and induces its proteasomal degradation. J Virol 81:3087–3096
3. Bensaude E, Turner JLE, Wakeley PR, Sweetman DA, Pardieu C, Drew TW, Wileman T, Powell PP (2004) Classical swine fever virus induces proinflammatory cytokines and tissue factor expression and inhibits apoptosis and interferon synthesis during the establishment of long-term infection of porcine vascular endothelial cells. J Gen Virol 85:1029–1037
4. Campos E, Revilla C, Chamorro S, Alvarez B, Ezquerra A, Dominguez J, Alonso F (2004) In vitro effect of classical swine fever virus on a porcine aortic endothelial cell line. Vet Res 35:625–633
5. Crow DS, Beyer EC, Paul DL, Kobe SS, Lau AF (1990) Phosphorylation of connexin43 gap junction protein in uninfected and Rous sarcoma virus-transformed mammalian fibroblasts. Mol Cell Biol 10:1754–1763
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