H3K9me3, H3K36me3, and H4K20me3 Expression Correlates with Patient Outcome in Esophageal Squamous Cell Carcinoma as Epigenetic Markers-Reference-Cited by-同舟云学术

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H3K9me3, H3K36me3, and H4K20me3 Expression Correlates with Patient Outcome in Esophageal Squamous Cell Carcinoma as Epigenetic Markers

Published:2019-02-20 Issue:8 Volume:64 Page:2147-2157
ISSN:0163-2116
Container-title:Digestive Diseases and Sciences
language:en
Short-container-title:Dig Dis Sci

Author:

Zhou Menghan,Li Yiping,Lin Shaofeng,Chen Yanping,Qian Yanyan,Zhao Zhujiang,Fan Hong^ORCID

Funder

National Natural Science Foundation of China

the Innovation Capability Development Project of Jiangsu Province

the Key Project of Cutting-edge Clinical Technology of Jiangsu Province

Jiangsu Provincial Department of Human Resources and Social Security

Publisher

Springer Science and Business Media LLC

Subject

Gastroenterology,Physiology

Link

http://link.springer.com/article/10.1007/s10620-019-05529-2/fulltext.html

Reference56 articles.

1. Bernard WS, Christopher PW. World cancer report. Lyon: International Agency for Research on Cancer; 2014.

2. Enzinger PC, Mayer RJ. Esophageal cancer. N Engl J Med. 2003;349:2241–2252.

3. Wong SH, Goode DL, Iwasaki M, et al. The H3K4-methyl epigenome regulates leukemia stem cell oncogenic potential. Cancer Cell. 2015;28:198–209.

4. Katoh H, Qin ZS, Liu R, et al. FOXP3 orchestrates H4K16 acetylation and H3K4 tri-methylation for activation of multiple genes through recruiting MOF and causing displacement of PLU-1. Mol Cell. 2011;44:770–784.

5. Zee BM, Levin RS, Xu B, LeRoy G, Wingreen NS, Garcia BA. In vivo residue-specific histone methylation dynamics. J Biol Chem. 2010;285:3341–3350.

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