Significance of IL-1RA Polymorphism in Iranian Patients with Inflammatory Bowel Disease-Reference-Cited by-同舟云学术

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Significance of IL-1RA Polymorphism in Iranian Patients with Inflammatory Bowel Disease

Published:2014-12-03 Issue:5 Volume:60 Page:1389-1395
ISSN:0163-2116
Container-title:Digestive Diseases and Sciences
language:en
Short-container-title:Dig Dis Sci

Author:

Daryani Nasser Ebrahimi,Sadr Maryam,Moossavi Shirin,Shahkarami Sepideh,Soltani Samaneh,Farhadi Elham,Rezaei Nima

Publisher

Springer Science and Business Media LLC

Subject

Gastroenterology,Physiology

Link

http://link.springer.com/content/pdf/10.1007/s10620-014-3457-z.pdf

Reference56 articles.

1. Abraham C, Cho JH. Inflammatory bowel disease. N Engl J Med. 2009;361:2066–2078.

2. Lopetuso LR, Chowdhry S, Pizarro TT. Opposing functions of classic and novel IL-1 family members in gut health and disease. Front Immunol. 2013;4:181.

3. Wolf SE, Debroy MA, Ikeda H, et al. Increased small bowel epithelial turnover in interleukin-1 receptor knockout mice. Ann Surg. 2000;232:42–45.

4. Shaw MH, Kamada N, Kim YG, Nunez G. Microbiota-induced IL-1beta, but not IL-6, is critical for the development of steady-state TH17 cells in the intestine. J Exp Med. 2012;209:251–258.

5. Chang J, Burkett PR, Borges CM, Kuchroo VK, Turka LA, Chang CH. MyD88 is essential to sustain mTOR activation necessary to promote T helper 17 cell proliferation by linking IL-1 and IL-23 signaling. Proc Natl Acad Sci USA. 2013;110:2270–2275.

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