During Thioacetamide-Induced Acute Liver Failure, the Proliferative Response of Hepatocytes to Thyroid Hormone Is Maintained, Indicating a Potential Therapeutic Approach to Toxin-Induced Liver Disease

Author:

Malik Raza,Saich Rebecca,Rahman Tony,Hodgson Humphrey

Publisher

Springer Science and Business Media LLC

Subject

Gastroenterology,Physiology

Reference24 articles.

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2. Malik R, Selden C, Hodgson H (2002) The Role of non-parenchymal cells in liver growth. Semin Cell Dev Biol 13(6):425–431

3. Ohmura T, Ledda-Columbano GM, Piga R, et al. (1996) Hepatocyte proliferation induced by a single dose of a peroxisome proliferator. Am J Pathol 148(3):815–824

4. Columbano A, Ledda-Columbano GM, Pibii M, et al. (1997) Increased expression of c-fos, c-jun and LRF-1 is not required for in vivo priming of hepatocytes by the mitogen TCPOBOP. Oncogene 14(7):857–863

5. Francavilla A, Carr B, Azzarone, et al. (1994) Hepatocyte proliferation and gene expression induced by triiodothyronine in vivo and in vitro. Hepatology 20(5):1237–1241

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