Reply to "The Probability Regarding the Tumor-promoter Role of Porphyromonas gingivalis for the Gastric Carcinogenesis"
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Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s10620-024-08459-w.pdf
Reference7 articles.
1. Lo C-H, Kwon S, Wang L et al. Periodontal disease, tooth loss, and risk of oesophageal and gastric adenocarcinoma: a prospective study. Gut 2021;70:620–621.
2. Oriuchi M, Lee S, Uno K et al. Porphyromonas gingivalis Lipopolysaccharide Damages Mucosal Barrier to Promote Gastritis-Associated Carcinogenesis. Dig Dis Sci 2024;69:95–111. https://doi.org/10.1007/s10620-023-08142-6
3. Li C, Yu J, Gong Z. Probability regarding the tumor-promoter role of Porphyromonas gingivalis for the gastric carcinogenesis. Comment on “Porphyromonas gingivalis lipopolysaccharide damages mucosal barrier to promote gastritis-associated carcinogenesis”. Dig Dis Sci 2024. https://doi.org/10.1007/s10620-024-08357-1
4. Tari A, Kitadai Y, Sumii M, et al. Basis of decreased risk of gastric cancer in severe atrophic gastritis with eradication of Helicobacter pylori. Dig Dis Sci 2007;52(1):232–239. https://doi.org/10.1007/s10620-006-9411-y
5. Sekine H, Iijima K, Koike T et al. Regional differences in the recovery of gastric acid secretion after Helicobacter pylori eradication: evaluations with Congo red chromoendoscopy. Gastrointest Endosc 2006;64:678–685.
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