A Role of Cell Apoptosis in Lipopolysaccharide (LPS)-induced Nonlethal Liver Injury in d-galactosamine (d-GalN)-sensitized Rats-Reference-Cited by-同舟云学术

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A Role of Cell Apoptosis in Lipopolysaccharide (LPS)-induced Nonlethal Liver Injury in d-galactosamine (d-GalN)-sensitized Rats

Published:2007-10-13 Issue:5 Volume:53 Page:1316-1324
ISSN:0163-2116
Container-title:Digestive Diseases and Sciences
language:en
Short-container-title:Dig Dis Sci

Author:

Liu Liang-Ming,Zhang Ji-Xiang,Luo Jie,Guo Hong-Xing,Deng Huan,Chen Jian-Yong,Sun Sui-Lin

Publisher

Springer Science and Business Media LLC

Subject

Gastroenterology,Physiology

Link

http://link.springer.com/content/pdf/10.1007/s10620-007-9994-y.pdf

Reference38 articles.

1. Morrison DC, Danner RL, Dinarello CA et al (1994) Bacterial endotoxins and pathogenesis of Gram-negative infections: current status and future direction. J Endotoxin Res 1:71–83

2. Thirunavukkarasu C, Uemura T, Wang LF et al (2005) Normal rat hepatic stellate cells respond to endotoxin in LBP-independent manner to produce inhibitor(s) of DNA synthesis in hepatocytes. J Cell Physiol 204:654–665

3. Wang P, Chaudry IH (1996) Mechanism of hepatocellular dysfunction during hyperdynamic sepsis. Am J Physiol Regulatory Integrative Comp Physiol 270: R927–R938

4. Bone RC, Balk RA, Cerra FB et al (1992) Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. The ACCP/SCCM consensus conference committee. American college of chest physicians/society of critical care medicine. Chest 101:1644–1655

5. Galanos C, Freudenburg MA, Reutter W (1979) Galactosamine-induced sensitization to the lethal effect of endotoxin. Proc Natl Acad Sci USA 76:5939–5943

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