Local Pentraxin-2 Deficit Is a Feature of Intestinal Fibrosis in Crohn’s Disease
Author:
Funder
Genentech
Publisher
Springer Science and Business Media LLC
Subject
Gastroenterology,Physiology
Link
https://link.springer.com/content/pdf/10.1007/s10620-023-07909-1.pdf
Reference12 articles.
1. Cosnes J, Cattan S, Blain A et al. Long-term evolution of disease behavior of Crohn’s disease. Inflamm Bowel Dis 2002;8:244–250.
2. Rieder F, Fiocchi C, Rogler G. Mechanisms, management, and treatment of fibrosis in patients with inflammatory bowel diseases. Gastroenterology 2017;152:340-350.e6.
3. Latella G, Di Gregorio J, Flati V et al. Mechanisms of initiation and progression of intestinal fibrosis in IBD. Scand J Gastroenterol 2015;50:53–65.
4. Louis E, Collard A, Oger AF et al. Behaviour of Crohn’s disease according to the Vienna classification: changing pattern over the course of the disease. Gut 2001;49:777–782.
5. Nakagawa N, Barron L, Gomez IG et al. Pentraxin-2 suppresses c-Jun/AP-1 signaling to inhibit progressive fibrotic disease. JCI Insight 2016;1:e87446.
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