A Comment on “Enteroendocrine and Neuronal Mechanisms in Pathophysiology of Acute Infectious Diarrhea” by Camilleri, Nullens and Nelsen
Author:
Publisher
Springer Science and Business Media LLC
Subject
Gastroenterology,Physiology
Link
http://link.springer.com/content/pdf/10.1007/s10620-012-2174-8.pdf
Reference13 articles.
1. Lucas ML. Diarrhoeal disease through enterocyte secretion: a doctrine untroubled by proof. Exp Physiol. 2010;95:479–484.
2. Lucas ML, Thom MMM, Bradley JM, O’Reilly NF, McIlvenny TJ, Nelson YB. Escherichia coli heat stable (STa) enterotoxin and the upper small intestine: lack of evidence in vivo for net fluid secretion. J Mem Biol. 2005;206:29–42.
3. Lucas ML. Amendments to the theory underlying Ussing chamber data of chloride ion secretion after bacterial enterotoxin exposure. J Theor Biol. 2005;234:21–37.
4. Lucas ML, Duncan NW, O’Reilly NF, McIlvenny TJ, Nelson YB. Lack of remote vagally-mediated restoration of Escherichia coli enterotoxin (STa) inhibited fluid absorption from rat proximal jejunum. Neurogastroenterol Motil. 2008;20:532–538.
5. Eklund S, Jodal M, Lundgren O. The enteric nervous system participates in the secretory response to the heat stable enterotoxins of Escherichia coli in rats and cats. Neuroscience. 1985;14:673–681.
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1. An alternative explanation for the occurrence of short circuit current increases in the small intestine following challenge by bacterial enterotoxins;Medical Hypotheses;2013-10
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