A COL1A1 Promoter-Controlled Expression of TGF-β Soluble Receptor Inhibits Hepatic Fibrosis Without Triggering Autoimmune Responses-Reference-Cited by-同舟云学术

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A COL1A1 Promoter-Controlled Expression of TGF-β Soluble Receptor Inhibits Hepatic Fibrosis Without Triggering Autoimmune Responses

Published:2018-06-23 Issue:10 Volume:63 Page:2662-2672
ISSN:0163-2116
Container-title:Digestive Diseases and Sciences
language:en
Short-container-title:Dig Dis Sci

Author:

Zhang Shouhua,Gong Yuanqi,Xiao Juhua,Chai Yong,Lei Jun,Huang Hui,Xiang Tianxin,Shen Wei

Funder

National Natural Science Foundation of China

Education Department Scientific Research Foundation of Jiangxi Province

Publisher

Springer Science and Business Media LLC

Subject

Gastroenterology,Physiology

Link

http://link.springer.com/article/10.1007/s10620-018-5168-3/fulltext.html

Reference38 articles.

1. Gressner AM, Weiskirchen R, Breitkopf K, Dooley S. Roles of TGF-beta in hepatic fibrosis. Front Biosci. 2002;7:d793–d807.

2. Friedman SL. Molecular regulation of hepatic fibrosis, an integrated cellular response to tissue injury. J Biol Chem. 2000;275:2247–2250.

3. Castilla A, Prieto J, Fausto N. Transforming growth factors β1 and α in chronic liver disease Effects of interferon alfa therapy. N Eng J Med. 1991;324:933–940.

4. Hellerbrand C, Stefanovic B, Giordano F, Burchardt ER, Brenner DA. The role of TGFβ1 in initiating hepatic stellate cell activation in vivo. J Hepatol. 1999;30:77–87.

5. Friedman SL. Evolving challenges in hepatic fibrosis. Nat Rev Gastroenterol Hepatol. 2010;7:425–436.

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