Cationic amino acid transporters and their modulation by nitric oxide in cardiac muscle cells
Author:
Funder
National Heart, Lung, and Blood Institute
Publisher
Springer Science and Business Media LLC
Subject
Molecular Biology,Structural Biology,Biophysics
Link
https://link.springer.com/content/pdf/10.1007/s12551-021-00870-1.pdf
Reference56 articles.
1. Al-Saleh EA, Wheeler KP (1982) Transport of neutral amino acids by human erythrocytes. Biochim Biophys Acta 684:157–171. https://doi.org/10.1016/0005-2736(82)90001-3
2. Assreuy J, Cunha FQ, Liew FY, Moncada S (1993) Feedback inhibition of nitric oxide synthase activity by nitric oxide. Br J Pharmacol 108:833–837. https://doi.org/10.1111/j.1476-5381.1993.tb12886.x
3. Balligand JL, Kelly RA, Marsden PA, Smith TW, Michel T (1993) Control of cardiac muscle cell function by an endogenous nitric oxide signaling system. Proc Natl Acad Sci USA 90:347–351. https://doi.org/10.1073/pnas.90.1.347
4. Beckman JS, Koppenol WH (1996) Nitric oxide, superoxide, and peroxynitrite: the good, the bad, and ugly. Am J Physiol Cell Physiol 271:C1424–C1437. https://doi.org/10.1152/ajpcell.1996.271.5.C1424
5. Beyer SR, Mallmann RT, Jaenecke I, Habermeier A, Boissel J-P, Closs EI (2013) Identification of cysteine residues in human cationic amino acid transporter hCAT-2A that are targets for inhibition by N-ethylmaleimide. J Biol Chem 288:30411–30419. https://doi.org/10.1074/jbc.M113.490698
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