Characterization of K562 cells: uncovering novel chromosomes, assessing transferrin receptor expression, and probing pharmacological therapies-Reference-Cited by-同舟云学术

Characterization of K562 cells: uncovering novel chromosomes, assessing transferrin receptor expression, and probing pharmacological therapies

Published:2023-08-14 Issue:9 Volume:80 Page:
ISSN:1420-682X
Container-title:Cellular and Molecular Life Sciences
language:en
Short-container-title:Cell. Mol. Life Sci.

Author:

Karagiannis Tom C.^ORCID,Wall Meaghan,Ververis Katherine,Pitsillou Eleni,Tortorella Stephanie M.,Wood Peter A.,Rafehi Haloom,Khurana Ishant,Maxwell Scott S.,Hung Andrew,Vongsvivut Jitraporn,El-Osta Assam

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Cellular and Molecular Neuroscience,Pharmacology,Molecular Biology,Molecular Medicine

Link

https://link.springer.com/content/pdf/10.1007/s00018-023-04905-6.pdf

Reference65 articles.

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3. Lee SM, Bae JH, Kim MJ et al (2007) Bcr-Abl-independent imatinib-resistant K562 cells show aberrant protein acetylation and increased sensitivity to histone deacetylase inhibitors. J Pharmacol Exp Ther 322(3):1084–1092

4. Zhang B, Strauss AC, Chu S et al (2010) Effective targeting of quiescent chronic myelogenous leukemia stem cells by histone deacetylase inhibitors in combination with imatinib mesylate. Cancer Cell 17(5):427–442

5. Druker BJ, Tamura S, Buchdunger E et al (1996) Effects of a selective inhibitor of the Abl tyrosine kinase on the growth of Bcr-Abl positive cells. Nat Med 2(5):561–566

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