The role of metabolic reprogramming in tubular epithelial cells during the progression of acute kidney injury
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Cellular and Molecular Neuroscience,Pharmacology,Molecular Biology,Molecular Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00018-021-03892-w.pdf
Reference97 articles.
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2. Hoste EAJ, Kellum JA, Selby NM, Zarbock A, Palevsky PM, Bagshaw SM, Goldstein SL, Cerdá J, Chawla LS (2018) Global epidemiology and outcomes of acute kidney injury. Nat Rev Nephrol 14(10):607–625. https://doi.org/10.1038/s41581-018-0052-0
3. Bhargava P, Schnellmann RG (2017) Mitochondrial energetics in the kidney. Nat Rev Nephrol 13(10):629–646. https://doi.org/10.1038/nrneph.2017.107
4. Scholz H, Boivin FJ, Schmidt-Ott KM, Bachmann S, Eckardt KU, Scholl UI, Persson PB (2021) Kidney physiology and susceptibility to acute kidney injury: implications for renoprotection. Nat Rev Nephrol 17(5):335–349. https://doi.org/10.1038/s41581-021-00394-7
5. Isaka Y, Kimura T, Takabatake Y (2011) The protective role of autophagy against aging and acute ischemic injury in kidney proximal tubular cells. Autophagy 7(9):1085–1087. https://doi.org/10.4161/auto.7.9.16465
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