BACE2 suppression promotes β-cell survival and function in a model of type 2 diabetes induced by human islet amyloid polypeptide overexpression-Reference-Cited by-同舟云学术

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BACE2 suppression promotes β-cell survival and function in a model of type 2 diabetes induced by human islet amyloid polypeptide overexpression

Published:2017-03-23 Issue:15 Volume:74 Page:2827-2838
ISSN:1420-682X
Container-title:Cellular and Molecular Life Sciences
language:en
Short-container-title:Cell. Mol. Life Sci.

Author:

Alcarraz-Vizán Gema,Castaño Carlos,Visa Montse,Montane Joel,Servitja Joan-Marc,Novials Anna

Funder

Instituto de Salud Carlos III

Departament d’Innovació, Universitats i Empresa, Generalitat de Catalunya

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Cellular and Molecular Neuroscience,Pharmacology,Molecular Biology,Molecular Medicine

Link

http://link.springer.com/article/10.1007/s00018-017-2505-1/fulltext.html

Reference48 articles.

1. Saunders A, Kim T-W, Tanzi R (1999) BACE maps to chromosome 11 and a BACE homolog, BACE2, reside in the obligate Down Syndrome region of chromosome 21. Science 286:1255a

2. Sun X, Wang Y, Qing H et al (2005) Distinct transcriptional regulation and function of the human BACE2 and BACE1 genes. FASEB J 19:739–749. doi: 10.1096/fj.04-3426com

3. Lahiri D, Maloney B, Ge Y (2006) Functional domains of the BACE1 and BACE2 promoters and mechanisms of transcriptional suppression of the BACE2 promoter in normal neuronal cells. J Mol Neurosci 29:65–80. doi: 10.1385/JMN/29

4. Sinha S, Anderson JP, Barbour R et al (1999) Purification and cloning of amyloid precursor protein beta-secretase from human brain. Nature 402:537–540. doi: 10.1038/990114

5. Yan R, Bienkowski MJ, Shuck ME et al (1999) Membrane-anchored aspartyl protease with Alzheimer’s disease beta-secretase activity. Nature 402:533–537. doi: 10.1038/990107

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