MPZL1 suppresses the cancer stem-like properties of lung cancer through β-catenin/TCF4 signaling
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s10142-023-01232-8.pdf
Reference28 articles.
1. Beigbeder A, Chartier FJM, Bisson N (2017) MPZL1 forms a signalling complex with GRB2 adaptor and PTPN11 phosphatase in HER2-positive breast cancer cells. Sci Rep 7:11514. https://doi.org/10.1038/s41598-017-11876-9
2. Chen W, Zheng R, Baade PD et al (2016) Cancer statistics in China, 2015. CA Cancer J Clin 66:115–132. https://doi.org/10.3322/caac.21338
3. Dang HH, Ta HDK, Nguyen TTT et al (2022) Prospective role and immunotherapeutic targets of sideroflexin protein family in lung adenocarcinoma: evidence from bioinformatics validation. Funct Integr Genomics 22:1057–1072. https://doi.org/10.1007/s10142-022-00883-3
4. Fang L, Zhu Q, Neuenschwander M et al (2016) A small-molecule antagonist of the β-catenin/tcf4 interaction blocks the self-renewal of cancer stem cells and suppresses tumorigenesis. Cancer Res 76:891–901. https://doi.org/10.1158/0008-5472.Can-15-1519
5. Feng J, Ouyang H, Wang J et al (2022) MPZL1 upregulation promotes tumor metastasis and correlates with unfavorable prognosis in non-small cell lung cancer. Carcinogenesis 43:919–929. https://doi.org/10.1093/carcin/bgac055
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