CMTM 6 promotes the development of thyroid cancer by inhibiting NIS activity through activating the MAPK signaling pathway
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Publisher
Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s10142-024-01298-y.pdf
Reference28 articles.
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2. Chai W, Ye F, Zeng L, Li Y, Yang L (2019) HMGB1-mediated autophagy regulates sodium/iodide symporter protein degradation in thyroid cancer cells. J Exp Clin Cancer Res 38:325. https://doi.org/10.1186/s13046-019-1328-3
3. Cunha LL, Marcello MA, Rocha-Santos V, Ward LS (2017) Immunotherapy against endocrine malignancies: immune checkpoint inhibitors lead the way. Endocr Relat Cancer 24:T261–T281. https://doi.org/10.1530/ERC-17-0222
4. Hong CM, Ahn BC (2017) Redifferentiation of Radioiodine Refractory Differentiated Thyroid Cancer for Reapplication of I-131 Therapy. Front Endocrinol (lausanne) 8:260. https://doi.org/10.3389/fendo.2017.00260
5. Huang X, Xiang L, Wang B et al (2021) CMTM6 promotes migration, invasion, and EMT by interacting with and stabilizing vimentin in hepatocellular carcinoma cells. J Transl Med 19:120. https://doi.org/10.1186/s12967-021-02787-5
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