Uric acid concentration in saliva and its changes with the patients receiving treatment for hyperuricemia

Author:

Shibasaki Koichi,Kimura Masatoshi,Ikarashi Ryuichi,Yamaguchi Akira,Watanabe Takuya

Publisher

Springer Science and Business Media LLC

Subject

Clinical Biochemistry,Biochemistry,Endocrinology, Diabetes and Metabolism

Reference11 articles.

1. Anzai, N., & Endo, H. (2008). Urate transporter 1: URAT1, Organic ion transporter: SLC22. Hyperuricemia and Gout, 16, 62–66. (in Japanese).

2. Committee on Biochemical Constituents, Their Analytical Reagents, Japan Society of Clinical Chemistry. (1993). Recommended method for determination of uric acid in serum by HPLC. Rinsho Kagagu, 22, 300–307. (in Japanese).

3. Enomoto, A., Kimura, H., Chairoungdua, A., et al. (2002). Molecular identification of a renal urate-anion exchanger that regulates blood urate levels. Nature, 417, 447–455.

4. Hikita, M., & Hosoya, T. (2004). Metabolic syndrome and hyperuricemia. Shindan-to-Chiryo, 92, 2057–2061. (in Japanese).

5. Hirose, M., Fukuda, A., Yahata, S., Matsumoto, D., & Igarashi, S. (2006). Individual variation in salivary buffer capacity measured by checkbuff and relationship among salivary flow rate, pH, buffer capacity, phosphate ion, and protein concentrations in saliva. Journal of Dental Health, 56, 220–227. (in Japanese).

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