Identification of glucose-independent and reversible metabolic pathways associated with anti-proliferative effect of metformin in liver cancer cells
Author:
Funder
University Grants Commission
Saha Institute of Nuclear Physics, Department of Atomic Energy, Government of India
Science and Engineering Research Board
Publisher
Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s11306-024-02096-0.pdf
Reference74 articles.
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2. Alshawi, A., & Agius, L. (2019). Low metformin causes a more oxidized mitochondrial NADH/NAD redox state in hepatocytes and inhibits gluconeogenesis by a redox-independent mechanism. Journal of Biological Chemistry, 294(8), 2839–5691. https://doi.org/10.1074/JBC.RA118.006670
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4. Ascha, M. S., Hanouneh, I. A., Lopez, R., Tamimi, T. A. R., Feldstein, A. F., & Zein, N. N. (2010). The incidence and risk factors of hepatocellular carcinoma in patients with nonalcoholic steatohepatitis. Hepatology, 51(6), 1972–1978. https://doi.org/10.1002/HEP.23527
5. Blandino, G., Valerio, M., Cioce, M., Mori, F., Casadei, L., Pulito, C., Sacconi, A., Biagioni, F., Cortese, G., Galanti, S., Manetti, C., Citro, G., Muti, P., & Strano, S. (2012). Metformin elicits anticancer effects through the sequential modulation of DICER and c-MYC. Nature Communications, 3(1), 865. https://doi.org/10.1038/ncomms1859
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