CYP1A2 polymorphisms, occupational and environmental exposures and risk of bladder cancer

Author:

Pavanello Sofia,Mastrangelo Giuseppe,Placidi Donatella,Campagna Marcello,Pulliero Alessandra,Carta Angela,Arici Cecilia,Porru Stefano

Publisher

Springer Science and Business Media LLC

Subject

Epidemiology

Reference60 articles.

1. Landi MT, Sinha R, Lang NP, Kadlubar FF. Chapter 16. Human cytochrome P4501A2. In: Vineis P, Malats N, Lang M, d’Errico A, Caporaso N, Cuzick J, Moffetta P, editors. Metabolic polymorphisms and susceptibility to cancer. Lyon: IARC Scientific Publications, IARC; 1999. 148, p. 173–195.

2. Bartsch H, Nair U, Risch A, Rojas M, Wikman H, Alexandrov K. Genetic polymorphism of CYP genes, alone or in combination, as a risk modifier of tobacco-related cancers. Cancer Epidemiol Biomarkers Prev. 2000;9:3–28.

3. Butler MA, Lang NP, Young JF, Caporaso NE, Vineis P, Hayes RB, Teitel CH, Massengill JP, Lawsen MF, Kadlubar FF. Determination of CYP1A2 and NAT2 phenotypes in human populations by analysis of caffeine urinary metabolites. Pharmacogenetics. 1992;2:116–27.

4. Pavanello S, Simioli P, Lupi S, Gregorio P, Clonfero E. Exposure levels and cytochrome P450 1A2 activity, but not N-acetyltransferase, glutathione S-transferase (GST) M1 and T1, influence urinary mutagen excretion in smokers. Cancer Epidemiol Biomarkers Prev. 2002;11:998–1003.

5. Sesardic D, Boobis AR, Edwards RJ, Davies DS. A form of cytochrome P450 in man, orthologous to form d in the rat, catalyses the odeethylation of phenacetin and is inducible by cigarette smoking. Br J Clin Pharmacol. 1988;26:363–72.

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