A high level of hybrid dysgenesis in Drosophila: High thermosensitivity, dependence on DNA repair, and incomplete cytotype regulation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics,Molecular Biology
Link
http://link.springer.com/content/pdf/10.1007/BF00391752.pdf
Reference41 articles.
1. Bingham PM, Kidwell MG, Rubin GM (1982) The molecular basis of P-M hybrid dysgenesis: the role of the P element, a P strain specific transposon family. Cell 29:995–1004
2. Black DM, Jackson MS, Kidwell GM, Dover GA (1987) KP elements repress P-induced hybrid dysgenesis in D. melanogaster. EMBO J 6:4125–4135
3. Boyd JB, Setlow R (1976) Characterization of post-replication repair in mutagen-sensitive strains of Drosophila melanogaster. Genetics 84:507–526
4. Boyd JB, Golino M, Setlow R (1976) The mei-9 mutant of Drosophila melanogaster increases mutagen sensitivity and decreases excision repair. Genetics 84:527–544
5. Bregliano JC, Kidwell MG (1983) Hybrid dysgenesis determinants. In: Shapiro JA (ed) Mobile genetic elements. Academic Press, New York
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2. Involvement of DNA Repair Genes and System of Radiation-Induced Activation of Transposons in Formation of Transgenerational Effects;Frontiers in Genetics;2020-11-27
3. Whole genome sequencing in Drosophila virilis identifies Polyphemus, a recently activated Tc1-like transposon with a possible role in hybrid dysgenesis;Mobile DNA;2014
4. Diversity of mechanisms and functions of enzyme systems of DNA repair in Drosophila melanogaster;Russian Journal of Genetics;2006-04
5. Molecular cloning of mei-41, a gene that influences both somatic and germline chromosome metabolism of Drosophila melanogaster;Molecular and General Genetics MGG;1995-03
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