Multi-drug strategies are necessary to inhibit the synergistic mechanism causing tissue damage and organ failure in post infectious sequelae

Author:

Ginsburg Isaac

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Pharmacology,Immunology

Reference57 articles.

1. Baue, A. E. (1992). The horror autotoxicus and multiple-organ failure, Arch. Surg. 127, 1451–1462.

2. Baue, A. E. (1998). Systemic inflammatory response syndrome (SIRS), multiple organ dysfunction syndrome (MODS), multiple organ failure (MOF): are we winning the battle?, Shock 10, 79–89.

3. Baue, A. E., Berlot, A. and Gullo, A. (Eds) (1998). Sepsis and Organ Dysfunction: Epidemiology and Scoring Systems, Pathophysiology and Therapy. Springer Verlag, Milan.

4. Baird, B. R., Cherionis, J. et al. (1986). Oxygen metabolites and neutrophil elastase synergistically cause edematous injury in isolated rat lungs, J. Appl. Physiol. 161, 224–2229.

5. Dan, P., Nizan, D. et al. (1996). H2O2 degrades cell surface proteoglycans and exposes the cells to lysis by Phospholipase A2: A novel mechanism for cell damage in inflammatory processes, FEBS Letters 383, 57–78.

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