The HIV infection and immune activation: ‘To fight and burn’
Author:
Publisher
Springer Science and Business Media LLC
Subject
Infectious Diseases
Link
http://link.springer.com/content/pdf/10.1007/s11908-005-0050-9.pdf
Reference50 articles.
1. Borkow G, Leng Q, Weisman Z, et al.: Chronic immune activation associated with intestinal helminth infections results in impaired signal transduction and anergy. J Clin Invest 2000, 106:1053–1060.
2. Giorgi JV, Hultin LE, McKeating JA, et al.: Shorter survival in advanced human immunodeficiency virus type 1 infection is more closely associated with T lymphocyte activation than with plasma virus burden or virus chemokine coreceptor usage. J Infect Dis 1999, 179:859–870. This article is one of the key papers that first demonstrated the link between immune activation, immunopathology and disease progression with specific emphasis on the importance of the CD38 activation marker.
3. Bentwich Z, Kalinkovich A, Weisman Z, Grossman Z: Immune activation in the context of HIV infection. Clin Exp Immunol 1998, 111:1–2.
4. McCune JM: The dynamics of CD4+ T-cell depletion in HIV disease. Nature 2001, 410:974–979.
5. Hazenberg MD, Otto SA, Cohen Stuart JW, et al.: Increased cell division but not thymic dysfunction rapidly affects the T-cell receptor excision circle content of the naive T cell population in HIV-1 infection. Nat Med 2000, 6:1036–1042. T-cell-receptor excision circles content in healthy individuals is only indirectly related to thymic output, and in HIV-1 infection is mainly affected by immune activation. This paper is one of the first to strongly suggest that naïve CD4 T cell depletion can result from immune activation and not only from thymus atrophy.
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