Sepsis and Cerebral Dysfunction: BBB Damage, Neuroinflammation, Oxidative Stress, Apoptosis and Autophagy as Key Mediators and the Potential Therapeutic Approaches-Reference-Cited by-同舟云学术

Sepsis and Cerebral Dysfunction: BBB Damage, Neuroinflammation, Oxidative Stress, Apoptosis and Autophagy as Key Mediators and the Potential Therapeutic Approaches

Published:2020-09-02 Issue:2 Volume:39 Page:489-503
ISSN:1029-8428
Container-title:Neurotoxicity Research
language:en
Short-container-title:Neurotox Res

Author:

Gu Ming,Mei Xiang-Lin,Zhao Ya-Nan

Publisher

Springer Science and Business Media LLC

Subject

Toxicology,General Neuroscience

Link

https://link.springer.com/content/pdf/10.1007/s12640-020-00270-5.pdf

Reference114 articles.

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2. Agac D, Estrada LD, Maples R, Hooper LV, Farrar JD (2018) The beta2-adrenergic receptor controls inflammation by driving rapid IL-10 secretion. Brain Behav Immun 74:176–185. https://doi.org/10.1016/j.bbi.2018.09.004

3. Agapito Fonseca J, Gameiro J, Marques F, Lopes JA (2020) Timing of initiation of renal replacement therapy in sepsis-associated acute kidney injury. J Clin Med 9:9. https://doi.org/10.3390/jcm9051413

4. Barichello T et al (2007a) Antioxidant treatment prevented late memory impairment in an animal model of sepsis. Crit Care Med 35:2186–2190. https://doi.org/10.1097/01.ccm.0000281452.60683.96

5. Barichello T et al (2007b) Behavioral deficits in sepsis-surviving rats induced by cecal ligation and perforation. Braz J Med Biol Res 40:831–837. https://doi.org/10.1590/s0100-879x2007000600013

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