The Protective Effects of AT2R Agonist, CGP42112A, Against Angiotensin II-Induced Oxidative Stress and Inflammatory Response in Astrocytes: Role of AT2R/PP2A/NFκB/ROS Signaling-Reference-Cited by-同舟云学术

The Protective Effects of AT2R Agonist, CGP42112A, Against Angiotensin II-Induced Oxidative Stress and Inflammatory Response in Astrocytes: Role of AT2R/PP2A/NFκB/ROS Signaling

Published:2021-09-16 Issue:6 Volume:39 Page:1991-2006
ISSN:1029-8428
Container-title:Neurotoxicity Research
language:en
Short-container-title:Neurotox Res

Author:

Bhat Shahnawaz Ali,Fatima Zoya,Sood Anika,Shukla Rakesh,Hanif Kashif^ORCID

Funder

Department of Biotechnology , Ministry of Science and Technology

Publisher

Springer Science and Business Media LLC

Subject

Toxicology,General Neuroscience

Link

https://link.springer.com/content/pdf/10.1007/s12640-021-00403-4.pdf

Reference58 articles.

1. Barnham KJ, Masters CL, Bush AI (2004) Neurodegenerative diseases and oxidative stress. Nat Rev Drug Discov 3:205. https://doi.org/10.1038/nrd1330

2. Bhat SA, Goel R, Shukla R, Hanif K (2016) Angiotensin receptor blockade modulates NFkappaB and STAT3 signaling and inhibits glial activation and neuroinflammation better than angiotensin-converting enzyme inhibition. Mol Neurobiol 53:6950–6967. https://doi.org/10.1007/s12035-015-9584-5

3. Bhat SA, Goel R, Shukla R, Hanif K (2017) Platelet CD40L induces activation of astrocytes and microglia in hypertension. Brain Behav Immun 59:173–189. https://doi.org/10.1016/j.bbi.2016.09.021

4. Bhat SA, Goel R, Shukla S, Shukla R, Hanif K (2018a) Angiotensin receptor blockade by inhibiting glial activation promotes hippocampal neurogenesis via activation of Wnt/β-catenin signaling in hypertension. Mol Neurobiol 55:5282–5298. https://doi.org/10.1007/s12035-017-0754-5

5. Bhat SA, Sood A, Shukla R, Hanif K (2018b) AT2R activation prevents microglia pro-inflammatory activation in a NOX-dependent manner: inhibition of PKC activation and p47(phox) phosphorylation by PP2A. Mol Neurobiol 56:3005–3023. https://doi.org/10.1007/s12035-018-1272-9

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