The Dephosphorylation of p70S6 (Thr389) Kinase as a Marker of l-Glutamate-Induced Excitotoxicity Related to Diabetes Disturbances—an Unconventional In Vitro Model
Author:
Funder
Uniwersytet Medyczny im. Piastów Slaskich we Wroclawiu
Publisher
Springer Science and Business Media LLC
Subject
Toxicology,General Neuroscience
Link
http://link.springer.com/content/pdf/10.1007/s12640-019-00155-2.pdf
Reference54 articles.
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2. Basse AL, Dalbram E, Larsson L (2018) Skeletal muscle insulin sensitivity show circadian rhythmicity which is independent of exercise training status. Front Physiol 9. https://doi.org/10.3389/fphys.2018.01198
3. Chen M, Zheng H, Wei T, Wang D, Xia H et al (2016) High glucose-induced PC12 cell death by increasing glutamate production and decreasing methyl group metabolism. Biomed Res Int 2016. https://doi.org/10.1155/2016/4125731
4. Ci Y, Shi K, An J, , Yang Y, Hui K, Wu P., Shi L., Xu C. (2014) ROS inhibit autophagy by downregulating ULK1 mediated by the phosphorylation of p53 in selenite-treated NB4 cells. Cell Death Dis doi: https://doi.org/10.1038/cddis.2014.506, 5
5. Dada T (2017) Is glaucoma a neurodegeneration caused by central insulin resistance: diabetes type 4. J Curr Glaucoma Pract 11:77–79
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