Ethylmalonic Acid Induces Permeability Transition in Isolated Brain Mitochondria-Reference-Cited by-同舟云学术

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Ethylmalonic Acid Induces Permeability Transition in Isolated Brain Mitochondria

Published:2014-02-21 Issue:2 Volume:26 Page:168-178
ISSN:1029-8428
Container-title:Neurotoxicity Research
language:en
Short-container-title:Neurotox Res

Author:

Cecatto Cristiane,Amaral Alexandre Umpierrez,Leipnitz Guilhian,Castilho Roger Frigério,Wajner Moacir

Publisher

Springer Science and Business Media LLC

Subject

Toxicology,General Neuroscience

Link

http://link.springer.com/content/pdf/10.1007/s12640-014-9460-5.pdf

Reference67 articles.

1. Adam-Vizi V, Starkov AA (2010) Calcium and mitochondrial reactive oxygen species generation: how to read the facts. J Alzheimers Dis 20(Suppl 2):S413–426. doi: 10.3233/JAD-2010-100465

2. Akerman KE, Wikstrom MK (1976) Safranine as a probe of the mitochondrial membrane potential. FEBS Lett 68(2):191–197

3. Amaral AU, Cecatto C, Busanello EN, Ribeiro CA, Melo DR, Leipnitz G, Castilho RF, Wajner M (2012) Ethylmalonic acid impairs brain mitochondrial succinate and malate transport. Mol Genet Metab 105(1):84–90. doi: 10.1016/j.ymgme.2011.10.006

4. Amendt BA, Greene C, Sweetman L, Cloherty J, Shih V, Moon A, Teel L, Rhead WJ (1987) Short-chain acyl-coenzyme a dehydrogenase deficiency. Clinical and biochemical studies in two patients. J Clin Invest 79(5):1303–1309. doi: 10.1172/JCI112953

5. Baines CP, Kaiser RA, Purcell NH, Blair NS, Osinska H, Hambleton MA, Brunskill EW, Sayen MR, Gottlieb RA, Dorn GW, Robbins J, Molkentin JD (2005) Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death. Nature 434(7033):658–662. doi: 10.1038/nature03434

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1. Ethylmalonic acid impairs bioenergetics by disturbing succinate and glutamate oxidation and induces mitochondrial permeability transition pore opening in rat cerebellum;Journal of Neurochemistry;2021-04-28

2. Acute Strokelike Presentation and Long-term Evolution of Diffusion Restriction Pattern in Ethylmalonic Encephalopathy;Journal of Child Neurology;2021-04-26

3. Mitochondrial Dysfunction and Redox Homeostasis Impairment as Pathomechanisms of Brain Damage in Ethylmalonic Encephalopathy: Insights from Animal and Human Studies;Cellular and Molecular Neurobiology;2020-10-09

4. Impairment of mitochondrial bioenergetics and permeability transition induction caused by major long-chain fatty acids accumulating in VLCAD deficiency in skeletal muscle as potential pathomechanisms of myopathy;Toxicology in Vitro;2020-02

5. Neurological manifestations of organic acidurias;Nature Reviews Neurology;2019-03-26

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