A Panel of Autoantibodies Against Neural Proteins as Peripheral Biomarker for Pesticide-Induced Neurotoxicity-Reference-Cited by-同舟云学术

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A Panel of Autoantibodies Against Neural Proteins as Peripheral Biomarker for Pesticide-Induced Neurotoxicity

Published:2017-09-05 Issue:2 Volume:33 Page:316-336
ISSN:1029-8428
Container-title:Neurotoxicity Research
language:en
Short-container-title:Neurotox Res

Author:

El Rahman Heba Allah Abd,Salama Mohamed,Gad El-Hak Seham A,El-Harouny Mona A,ElKafrawy Passent,Abou-Donia Mohamed B.

Funder

Internationalf Brain Research Organization/African Regional Chapter (IBRO/ARC)

International Society of Neurochemistry

Mansoura University Competitive Research Grants

Publisher

Springer Science and Business Media LLC

Subject

Toxicology,General Neuroscience

Link

http://link.springer.com/article/10.1007/s12640-017-9793-y/fulltext.html

Reference50 articles.

1. Abdel-Rahman A, Shetty AK, Abou-Donia AK (2001) Subchronic dermal application of N,N-diethyl m-toluidamide (DEET) and permethrin to adults rats, alone or in combination, causes diffuse neuronl cell death and cytoskeletal abnormalities in the cerebral cortex and hippocampus, and Purknije neuron loss in the cerebellum. Exp Neurol 172:153–171

2. Abdel-Rahman AA, Shetty AK, Abou-Donia MB (2002a) Disruption of blood brain barrier and neuronal cell death in cingulate cortex, dentate gyrus, thalamus, and hypothalamus in a rat model of Gulf-War syndrome. Neurob Disease 10:306–326

3. Abdel-Rahman AA, Shetty AK, Abou-Donia MB (2002b) Acute exposure to sarin increases blood brain barrier permeability and induces neuropathological changes in the rat brain: dose response relationship. Neuroscience 113:721–741

4. Abdel-Rahman AA, Goldstein LB, Bulman SL, Khan WA, El Masry EM, Abou-Donia MB (2004a) Neuronal degeneration and neurobehavioral deficits following dermal exposure to malathion, DEET, and permethrin, alone and in combination. J Toxicol Environ Health 67:331–356

5. Abdel-Rahman A, Shetty AK, Abou-Donia SM, El-Masry EM, Abou-Donia MB (2004b) Stress and combined chemical exposure produce neurochemical and neuropathological alterations in cerebral cortex, hippocampus, and cerebellum. J Toxicol Environ Health 67:163–192

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