MiR-27a-3p/miR-27b-3p Promotes Neurofibromatosis Type 1 via Targeting of NF1
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s12031-020-01779-2.pdf
Reference22 articles.
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2. Barkan B, Starinsky S, Friedman E, Stein R, Kloog Y (2006) The Ras inhibitor farnesylthiosalicylic acid as a potential therapy for neurofibromatosis type 1 Clinical cancer research : an official journal of the American Association for Cancer Research 12:5533–5542. https://doi.org/10.1158/1078-0432.Ccr-06-0792
3. Cappione AJ, French BL, Skuse GR (1997) A potential role for NF1 mRNA editing in the pathogenesis of NF1 tumors. Am J Hum Genet 60:305–312
4. Cimino PJ, Gutmann DH (2018) Neurofibromatosis type 1. Handb Clin Neurol 148:799–811. https://doi.org/10.1016/b978-0-444-64076-5.00051-x
5. Frassanito MA et al (2019) Bone marrow fibroblasts overexpress miR-27b and miR-214 in step with multiple myeloma progression, dependent on tumour cell-derived exosomes. J Pathol 247:241–253. https://doi.org/10.1002/path.5187
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