Different Activation of IL-10 in the Hippocampus and Prefrontal Cortex During Neurodegeneration Caused by Trimethyltin Chloride
Author:
Funder
State task of ITEB RAS
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s12031-020-01682-w.pdf
Reference19 articles.
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2. de Bilbao F, Arsenijevic D, Moll T, Garcia-Gabay I, Vallet P, Langhans W, Giannakopoulos P (2009) In vivo over-expression of interleukin-10 increases resistance to focal brain ischemia in mice. J Neurochem. 110:12–22. https://doi.org/10.1111/j.1471-4159.2009.06098.x
3. Garcia JM, Stillings SA, Leclerc JL, Phillips H, Edwards NJ, Robicsek SA, Hoh BL, Blackburn S, Doré S (2017) Role of interleukin-10 in acute brain injuries. Front Neurol 8:244. https://doi.org/10.3389/fneur.2017.00244
4. Geloso MC, Corvino V, Michetti F (2011) Trimethyltin-induced hippocampal degeneration as a tool to investigate neurodegenerative processes. Neurochem Int 58:729–738. https://doi.org/10.1016/j.neuint.2011.03.009
5. Harvey D, Smith R, English K, Mahon B, Commins S (2006) Interleukin-10 (IL-10) but not lipopolysaccharide (LPS) produces increased motor activity and abnormal exploratory patterns while impairing spatial learning in Balb/c mice. Physiol Behav 87:842–847. https://doi.org/10.1016/j.physbeh.2006.03.002
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