Glutamate Neonatal Excitotoxicity Modifies VEGF-A, VEGF-B, VEGFR-1 and VEGFR-2 Protein Expression Profiles During Postnatal Development of the Cerebral Cortex and Hippocampus of Male Rats-Reference-Cited by-同舟云学术

Glutamate Neonatal Excitotoxicity Modifies VEGF-A, VEGF-B, VEGFR-1 and VEGFR-2 Protein Expression Profiles During Postnatal Development of the Cerebral Cortex and Hippocampus of Male Rats

Published:2017-07-29 Issue:1 Volume:63 Page:17-27
ISSN:0895-8696
Container-title:Journal of Molecular Neuroscience
language:en
Short-container-title:J Mol Neurosci

Author:

Castañeda-Cabral Jose Luis,Beas-Zarate Carlos,Gudiño-Cabrera Graciela,Ureña-Guerrero Monica E.

Funder

Consejo Nacional de Ciencia y Tecnología

Universitario of Guadalajara

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Medicine

Link

http://link.springer.com/article/10.1007/s12031-017-0952-7/fulltext.html

Reference59 articles.

1. Beas-Zarate C, Rivera-Huizar SV, Martinez-Contreras A, Feria-Velasco A, Armendariz-Borunda J (2001) Changes in NMDA-receptor gene expression are associated with neurotoxicity induced neonatally by glutamate in the rat brain. Neurochem Int 39(1):1–10. doi: 10.1016/S0197-0186(01)00008-0

2. Beas-Zarate C, Ureña-Guerrero ME, Flores-Soto M, Armendariz-Borunda J, Ortuño-Sahagun D (2007) The expression and binding of kainate receptors is modified in different brain regions by glutamate neurotoxicity during postnatal rat development. Int J Dev Neurosci 25(1):53–61. doi: 10.1016/j.ijdevneu.2006.10.001

3. Brockington A, Lewis C, Wharton S, Shaw PJ (2004) Vascular endothelial growth factor and the nervous system. Neuropathol Appl Neurobiol 30(5):427–446. doi: 10.1111/j.1365-2990.2004.00600.x

4. Bry M, Kivelä R, Leppänen VM, Alitalo K (2014) Vascular endothelial growth factor-B in physiology and disease. Physiol Rev 94(3):779–794. doi: 10.1152/physrev.00028.2013

5. Chaparro-Huerta V, Rivera-Cervantes MC, Torres-Mendoza BM, Beas-Zarate C (2002) Neuronal death and tumor necrosis factor-alpha response to glutamate-induced excitotoxicity in the cerebral cortex of neonatal rats. Neurosci Lett 333(2):95–98. doi: 10.1016/S0304-3940(02)01006-6

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